Prandota J., Iwańczak F., Pytrus T.Different variants of position and length of colon transversum as the cause of recurrent abdominalgia and chronic constipation in 12 adolescent children.Pol. Merkuriusz Lek., 2003, 15, 47-50.
Prandota J.Urinary tract diseases revealed after DTP vaccination in infants and young children. Cytokine irregularities and down-regulation of cytochrome P-450 enzymes induced by the vaccine may uncover latent diseases in genetically predisposed subjects.Am. J. Ther., 2004, 11, 344-353.
Prandota J.Limitations in the clinical usefulness of a single-dose pharmacokinetic studies of drugs and a Bayesian approach for estimation of kinetic parameters. Am. J. Ther., 2004, 11, 295-301.
Prandota J.Possible pathomechanisms of sudden infant death syndrome. Key role of chronic hypoxia, infection/inflammation states, cytokine irregularities, and metabolic trauma in genetically predisposed infants. Am. J. Ther., 2004, 11, 517-546.
Prandota J. Important role of proinflammatory cytokines/other endogenous substances in drug-induced hepatoxicity. Depression of drug metabolism during infections/ inflammation states, and genetic polymorphisms of drug-metabolizing enzymes/ cytokines may markedly contribute to this pathology. Am. J. Ther., 2005, 12 , 254-261.
Prandota J. Recurrent headache as the main symptom of acquired cerebral toxoplasmosis in non-human immunodeficiency virus-Infected subjects with no lymphadenopathy. The parasite may be responsible for the neurogenic inflammation postulated as a cause of different types of headaches Am. J. Ther., 2007, 14, 63-105.
Prandota J.Mollaret meningitis may be caused by reactivation of latent cerebral toxoplasmosis.Int. J. Neurosci., 2009, 19, 1655-1692.
Prandota J.The importance of Toxoplasma gondii infection in disease presenting with headaches. Headaches and aseptic meningitis may be manifestations of the Jarisch-Herxheimer reaction.Int. J. Neurosci., 2009, 119, 2144-2182.
Prandota J. Advances of molecular clinical pharmacology in gastroenterology and hepatology.Am. J. Ther., 2010, 17, 137-162.
Prandota J.Neuropathological changes and clinical features of autism spectrum disorder participants are similar to the reported in congenital and acquired chronic cerebral toxoplasmosis in humans and mice.
Res Autism Spectr Disord, 2010, 4, 103-118.
Prandota J. Autism spectrum disorders may be due to cerebral toxoplasmosis associated with chronic neuroinflammation causing persistent hypercytokinemia that resulted in an increased lipid peroxidation, oxidative stress, and depressed metabolism of endogenous and exogenous substances.Res Autism Spectr Disord, 2010, 4, 119-155.
Prandota J. Migraine associated with patent foramen ovale may be caused by reactivation of cerebral toxoplasmosis triggered by arterial blood oxygen desaturation. Int J Neurosci, 2010, 120, 81-87.
Prandota J.Metabolic, immune, epigenetic, endocrine and phenotypic abnormalities found in individuals with autism spectrum disorders, Down syndrome and Alzheimer disease may be caused by congenital and/or acquired chronic cerebral toxoplasmosis. Res Autism Spectr Disord, 2011, 5, 14-59.
Prandota J. Idiopathic intracranial hypertension may be caused by reactivation of latent cerebral toxoplasmosis. Effect of drugs and biologic substances. In: Gemma C (ed.), Neuroinflammation. Pathogenesis, Mechanisms and Management, Nova Science Publishers, New York 2012, 273-336.
Prandota J. Idiopathic intracranial hypertension may be caused by reactivation of latent cerebral toxoplasmosis. Effect of various diseases and clinical states. In: Gemma C (ed.), Neuroinflammation. Pathogenesis, Mechanisms and Management, Nova Science Publishers, New York 2012, pp. 337-343.
Prandota J. Transient beneficial effects of fever on behavior of children with autistic spectrum disorders may be caused by T. gondii stage differentiation during latent cerebral toxoplasmosis, associated with the changes in immune responses of the host In: Gemma C (ed.), Neuroinflammation. Pathogenesis, Mechanisms and Management, Nova Science Publishers, New York 2012, pp. 399-421.
Prandota J. Rhesus-associated glycoprotein (RhAG) phenotype of the red blood cells modulates T. gondii infection-associated psychomotor performance reaction times and changes in the human personality profile. Impaired function of the CO2, AQP1, and AQP4 gas channels may cause hypoxia and thus enhance neuroinflammation in autistic individuals. In: Gemma C (ed.), Neuroinflammation. Pathogenesis, Mechanisms and Management, Nova Science Publishers, New York 2012, pp. 422-446.
Prandota J. Increased generation of antibodies and autoantibodies directed against brain proteins in patients with autism and their families may be caused by T. gondii infection. Maternal and fetal microchimerisms probably play an important role in these processes acting as a “Trojan horse” in dissemination of the parasite. In: Gemma C (ed.), Neuroinflammation. Pathogenesis, Mechanisms and Management, Nova Science Publishers, New York 2012, pp. 447-638.
Prandota J. Gastrointestinal tract abnormalities in autism, inflammatory bowel disease and many other clinical entities may be due to T. gondii infection Scientific Reports, 2012, 1, 4, doi.org/10.4172/scientificreports.256.
Prandota J. T. gondii infection acquired prenatally and/or after birth may be responsible for development of both type 1 and 2 diabetes mellitus. J Diabetes & Metabolism, 2013.
Prandota J.Possible link between T. gondii and the anosmia associated with neurodegenerative diseases.Am J Alzheimer’s Dis & Other Dement, 2014, 29, 205-214.
Prandota J, Gryglas A, Fuglewicz A, Żesławska-Faleńczyk A, Ujma-Czapska B, Szenborn L, Mierzwa L.Recurrent headaches may be caused by T. gondii infection. World J Clin Pediatr, 2014, 3, 59-68.
Flegr J, Prandota J, Sovičková M, Israili ZHToxoplasmosis – a global threat. Correlation of latent toxoplasmosis with specific disease burden in a set of 88 countries.PLoS ONE, 2014, 9(3):e90203.
Prandota J. Unexplained liver damage, development of cryptogenic liver cirrhosis, and alcoholic, and non-alcoholic steatohepatitis may be caused by latent T. gondii infection.OA Hepatology, 2014, Feb 10;2(1):2 (in press).
Prandota J, Elleboudy NAF, Ismail KA, Zaki OK, Shehata HH. Increased seroprevalence of chronic toxoplasmosis in autistic children: Special reference to the pathophysiology of IFN-gamma and NO overproduction.Int J Neurology Res, 2015, 1(3), 102-122.
Prandota J.Possible pivotal role of latent chronic T. gondii infection in the pathogenesis of atherosclerosis.J Cardiol Therapy, 2017, April, 4(2): 611-663.